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Seracare军团菌嗜肺军团菌阳性对照

广州健仑生物科技有限公司

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与此同时，该研究的结果也可以应用于人类机体其他组织，比如皮肤、肺部和抗原抗体等;揭示干细胞在人类肠道中的行为机制将是干细胞研究领域的一大飞跃，截至目前研究人员更多的是利用小鼠来进行研究或者是将干细胞置于自然环境中进行研究，这就扭曲了干细胞的正常行为表现;而这项研究中研究者利用开发的新型工具来揭示肠道干细胞的行为表现，这就可以明显增加研究人员对于肠癌发生的理解。
zui后，研究者Marnix Jansen博士说道，基于本文的研究，通过对来自肠癌患者的组织进行分析研究，我们就可以清楚揭示肠癌的发生机理，当然后期还需要进行更为深入的研究才能够为开发治疗肠癌患者的疾病以及改善其生存质量提供帮助。
尽管疗法已经得到了极大改善，但是两个成年急性髓性白血病(AML)患者中也仅有一人会生存下去，对于65岁以上的老年人而言，急性髓性白血病平均存活期不到一年;而研究者推测发病原因可能是白血病干细胞，在患者机体中白血病干细胞并不能*被清除，这些白血病干细胞就是引发患者病情复发zui终致死的原因。
近日，刊登在杂志 Cancer Research 上的一篇研究论文中，来自歌德大学( Goethe-Universitat Frankfurt am Main )的研究人员通过研究发现了急性髓性白血病干细胞的弱点，他们发现了一种名为5-脂氧合酶(5-LO)的酶类在急性髓性白血病干细胞的生存过程中扮演着重要角色。
此前研究人员已经发现了 5-LO 在炎性疾病比如哮喘症发病中的角色，研究者 Jessica Roos 教授表示，这项研究中我们发现，急性髓性白血病亚群中的白血病干细胞或许可以被5-LO抑制剂有效地选择性攻击，而且这种现象均可以在细胞培养基模型和白血病小鼠模型中观察到。
研究者表示，这项研究为开发新型的5-脂氧合酶抑制剂来抑制白血病的存货，从而为抑制并治疗急性髓性白血病提供了新的研究依据和线索。后期研究中研究人员还将通过深入研究来揭示5-脂氧合酶抑制剂在抑制白血病干细胞功能中的作用和机制。
科学家们早就知道，当细胞反复分裂时，就会发生染色体缺陷。随着时间的推移，这些缺陷就会导致癌症的发作。
现在，来自英国卡迪夫大学的邓肯·贝尔德教授和他的研究团队与来自明尼苏达大学的埃里克A·亨德里克森合作已经确定了，人类细胞为了生存所需的这些类型的缺陷的一个特定基因。
“我们发现，似乎是在进化过程中，可以驱动癌症的关键基因，” 卡迪夫大学的癌症与基因研究所的贝尔德教授说。“这是该基因的新作用，使其成为潜在的治疗靶点。”
随着细胞的分裂，它们的端粒——保护染色体末端免受损坏的DNA“帽”——缩短，使残余的染色体容易彼此粘在一起。
在正常细胞中，这种染色体的粘性是一个死亡的信号，该信号能够触发缺陷的细胞在清理过程中移动，并帮助清理它们的过程。
然而，恶性细胞，在某种程度上能够躲避这个清理过程。

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、食品安全、化妆品检测、药物滥用检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

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【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

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【腾讯  】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103室

At the same time, the results of this study can be applied to the human body's other tissues, such as skin, lungs and antigen antibodies; reveal the mechanisms of stem cell behavior in the human gut is the field of stem cell research will be a great leap forward, as of now researchers More is the use of mice to carry out research or put the stem cells in the natural environment for research, which distorted the normal behavior of stem cells; and researchers in this study using the development of new tools to reveal the intestinal stem cells Behavioral performance, which can significantly increase the researchers understanding of the occurrence of colorectal cancer.
Finally, the researcher Dr. Marnix Jansen says, research paper based on tissue from patients with colorectal cancer through the analysis, we can clearly reveal the mechanism of cancer, of course, the latter also need more in-depth study to be able to Development and treatment of diseases of patients with colorectal cancer and improve their quality of life to provide help.
Despite the dramatic improvement in therapies, only one in two adults with acute myeloid leukemia (AML) survives, with an average survival of less than one for acute myeloid leukemia in older adults over 65 years Year; and the researchers speculated that the pathogenesis may be leukemia stem cells, leukemia stem cells in the patient's body can not be compley removed, these leukemia stem cells is caused by the patient's condition eventually lethal cause of death.
Recently, in a research paper published in the International Cancer Research, researchers from Goethe-Universitat Frankfurt am Main discovered the weakness of acute myeloid leukemia stem cells through research that they found in a study entitled 5 - Lipoxygenase (5-LO) enzymes play an important role in the survival of acute myelogenous leukemia stem cells.
Previously researchers have found that 5-LO role in the pathogenesis of inflammatory diseases such as asthma in, the researchers Jessica Roos Professor said the study, we found that acute myeloid leukemia subgroup of leukemic stem cells might be 5- LO inhibitors effectively selectively attack, and this phenomenon can be observed in both cell culture and leukemia mouse models.
The researchers said the study provides new research evidence and clues for the suppression and treatment of acute myeloid leukemia in order to develop new 5-lipoxygenase inhibitors to inhibit the stock of leukemia. In the latter part of the study, researchers will further study to reveal the role and mechanism of 5-lipoxygenase inhibitors in inhibiting leukemia stem cell function.
Scientists have long known that chromosomal defects occur when cells repeatedly divide. Over time, these shortcomings can lead to the onset of cancer.
A now from Cardiff University's Professor Duncan Baird and his research team A · Eric Hendrickson from the cooperation with the University of Minnesota have identified, in order to survive in human cells required for these types of defects Specific gene.
"We found it to be a key gene that can drive cancer in the evolutionary process," said Professor Baird, a professor of cancer and genetics at Cardiff University. "This is a new role for the gene, making it a potential therapeutic target."
As the cells divide, their omeres - protecting the chromosome ends from damaged DNA "caps" - shorten the residual chromosomes to stick together easily.
In normal cells, the stickiness of this chromosome is a signal of death that triggers defective cells to move during the cleaning process and help cleanse them.
However, malignant cells, to some extent, are able to evade this cleanup process.